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Book Summary
Heart failure has been traditionally explained as a reduction in cardiac pumping
capacity, caused by problems in the blood. Till now the therapy prescribed has
been symptomatic, involving the use of diuretics, muscular stimulation and blood
vessel relaxants. But recent research has focussed on the circumstances leading
up to heart failure, which includes abnormal hemodynamics, atherosclerosis,
dietary and nutritional anomalies, obesity, immunity and other metabolic
disorders. This book brings together such research in one volume, including
molecular and cellular aspects of heart failure and its pathophysiology. It is a
significant contribution to the understanding and prevention of heart failure,
and will be welcomed by cardiologists, pharmacologists, biochemists, and
Heart failure has been traditionally explained as a reduction in cardiac pumping
capacity, caused by problems in the blood. Till now the therapy prescribed has
been symptomatic, involving the use of diuretics, muscular stimulation and blood
vessel relaxants. But recent research has focussed on the circumstances leading
up to heart failure, which includes abnormal hemodynamics, atherosclerosis,
dietary and nutritional anomalies, obesity, immunity and other metabolic
disorders. This book brings together such research in one volume, including
molecular and cellular aspects of heart failure and its pathophysiology. It is a
significant contribution to the understanding and prevention of heart failure,
and will be welcomed by cardiologists, pharmacologists, biochemists, and